KETOASIDOSIS DIABETIKUM PDF

Transcript of Ketoasidosis Diabetikum. Pengantar Tahun , pasien rawat inap KAD rata-rata lama menginap hari. Kelalaian. Download Citation on ResearchGate | Gambaran Klinis Ketoasidosis Diabetikum Anak | Penelitian ini dilakukan untuk menggambarkan profil klinis ketoasidosis. Penelitian ini dilakukan untuk menggambarkan profil klinis ketoasidosis diabetikum pada anak. Data dikumpulkan dari rekam medik gambaran klinis pasien.

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However, total body potassium deficit is usually present from urinary potassium losses due to osmotic diuresis and ketone excretion.

By continuing to use this website, you agree to their use. Kesalahan yang menyebabkan dosis insulin yang harus diberikan berkurang, terjadi pada pasien-pasien yang sakit dan menganggap jika mereka kurang makan atau menderita muntah-muntah, maka dosis insulinnya juga harus dikurangi.

Nonspecific hyperamylasemia and hyperlipasemia in diabetic ketoacidosis: Akibat defisiensi insulin yang lain adalah pemecahan lemak lipolisis menjadi asam — asam lemak bebas dan gliserol. Fatty acids, lipotoxicity and insulin secretion.

Archived PDF from the original on 24 March Frequency United States Currently, diabetic ketoacidosis DKA occurs less frequently in patients with known diabetes because of the introduction of diabetes educational programs in most diabetes clinics. If the patient is unable to eat, it is preferable to continue the intravenous insulin infusion and fluid replacement. Particularly important in this regard is captopril, an angiotensin converting enzyme inhibitor prescribed for the treatment of hypertension and diabetic nephropathy.

Penelitian ini dilakukan untuk menggambarkan profil klinis ketoasidosis diabetikum pada anak. DKA also occurs in pregnant women, either with preexisting diabetes or with diabetes diagnosed during pregnancy. There is often a particular underlying problem that has led to the DKA episode; this may be intercurrent illness pneumoniainfluenzagastroenteritisa urinary tract infectionpregnancyinadequate insulin administration e. Di negara maju dapat dikatakan penyebab utama kematian adalah infeksi, usia lanjut dan osmolaritas darah yang sangat tinggi.

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Untuk mengetahui manifestasi klinis dari Keto Asidosis d. Gunakan tingkat ini dalam hubungannya dengan kesenjangan anion untuk menilai derajat asidosis.

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Progressive rise of blood concentration of these acidic organic substances initially leads to a state of ketonemia. Several clinical studies have shown the potency and cost effectiveness of subcutaneous rapid-acting insulin analogs lispro or aspart in the management of patients with uncomplicated mild to moderate DKA Pada proses peleburan sulfida logam diubah menjadi oksida logam. Insulin menghambat pemecahan lemak sehingga menghentikan pembentukan senyawa-senyawa yang bersifat asam.

The worst prognosis is usually observed in patients who are older with severe intercurrent illnesses, eg, myocardial infarction, sepsis, or pneumonia, especially when they are treated outside an ICU. Apabila jumlah insulin berkurang, jumlah glukosa yang memasuki sel akan berkurang pula.

MAKALAH Keto Asidosis Diabetikum – PDF Free Download

Hyperglycemia presenting with occipital seizures. A low-dose insulin regimen has the diabegikum of not inducing severe hypoglycemia or hypokalemia, as may be observed with a high-dose insulin regimen.

Is treatment a factor? Can J Diabetes ; 42 Suppl 1: Diabetic acidosis with initial hypokalemia. Insulinopenia and elevated cortisol levels also lead to a shift from protein synthesis to proteolysis with ketoasjdosis increase in production of amino acids alanine and glutaminewhich further serve as substrates for gluconeogenesis 5 Kaji Kadar AGD setiap hari.

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Patients usually have normal body temperature or mild hypothermia regardless of presence of infection Pencegahan Dua faktor yang paling berperan dalam timbulnya KAD adalah terapi insulin yang tidak adekuat dan infeksi. Defisit volume cairan berhubungan dengan diuresis osmotik akibat hiperglikemia, pengeluaran cairan berlebihan: Mechanism of cerebral edema in children with diabetic ketoacidosis.

Keterampilan dalam menangani penyakit diabetes secara mandiri yang mencakup penyuntikan insulin dan pemeriksaan kadar glukosa darah harus dikaji dengan memastikan tidak terjadi kesalahan yang tidak disengaja pada pemberian insulin atau pemeriksaan kadar glukosa darah tersebut.

Archived from the original on 9 August Acta Riabetikum ; During follow up, blood should be drawn every h for determination of serum electrolytes, glucose, blood urea nitrogen, creatinine, osmolality, and venous pH. Correction of fluid loss makes the clinical picture clearer and may be sufficient to correct acidosis. Sagung Seto; ; hal. Hong Kong Medical Journal. Although DKA was a common problem in patients with diabetes who were treated with continuous subcutaneous insulin infusion SCII through diabetikkm infusion pumps, incidence of DKA became less frequent with the introduction of new pumps equipped with sensitive electronic alarm systems that alert users when the infusion catheter is blocked.

Kehilangan elektrolit merupakan kompensasi dari defisiensi insulin.

Pathogenesis of cerebral edema after treatment of diabetic ketoacidosis.